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All fields Title Creator / Publisher Keyword Year AND OR AND NOT All fields Title Creator / Publisher Keyword Year AND OR AND NOT All fields Title Creator / Publisher Keyword Year AND OR AND NOT All fields Title Creator / Publisher Keyword Year AND OR AND NOT All fields Title Creator / Publisher Keyword Year Sort by creator [A → Z]' creator [Z → A]' publishing year ↑ (asc)' publishing year ↓ (desc)' title [A → Z]' title [Z → A]' Simple Search Hits 1 – 4 of 4 1 Loss of functional connectivity is greater outside the default mode network in non-familial early-onset Alzheimer’s disease variants Lehmann, Manja; Madison, Cindee; Ghosh, Pia M.; Miller, Zachary A.; Greicius, Michael D.; Kramer, Joel H.; Coppola, Giovanni; Miller, Bruce L.; Jagust, William J.; Gorno-Tempini, Maria L.; Seeley, William W.; Rabinovici, Gil D.. - 2015 Abstract: The common and specific involvement of brain networks in clinical variants of Alzheimer’s disease (AD) is not well understood. We performed task-free (“resting-state”) functional imaging in 60 non-familial AD patients, including 20 early-onset AD (EOAD, age at onset <65 years, amnestic/dysexecutive deficits), 24 logopenic aphasia (lvPPA, language deficits) and 16 posterior cortical atrophy patients (PCA, visual deficits), as well as 60 healthy controls. Seed-based connectivity analyses were conducted to assess differences between groups in 3 default mode network (DMN) components (anterior, posterior and ventral) and four additional non-DMN networks: left and right executive-control, language and higher visual networks. Significant decreases in connectivity were found across AD variants compared with controls in the non-DMN networks. Within the DMN components, patients showed higher connectivity in the anterior DMN, in particular in lvPPA. No significant differences were found for the posterior and ventral DMN. Our findings suggest that loss of functional connectivity is greatest in networks outside the DMN in early-onset and non-amnestic AD variants, and may thus be a better biomarker in these patients. Keyword: Article URL: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4698410/ http://www.ncbi.nlm.nih.gov/pubmed/26242705 https://doi.org/10.1016/j.neurobiolaging.2015.06.029 BASE Hide details 2 Diverging patterns of amyloid deposition and hypometabolism in clinical variants of probable Alzheimer's disease Lehmann, Manja; Ghosh, Pia M.; Madison, Cindee. - : Oxford University Press, 2013 BASE Show details 3 Diverging patterns of amyloid deposition and hypometabolism in clinical variants of probable Alzheimer's disease Lehmann, Manja; Ghosh, Pia M.; Madison, Cindee. - : Oxford University Press, 2013 BASE Show details 4 Diverging patterns of amyloid deposition and hypometabolism in clinical variants of probable Alzheimer’s disease Lehmann, Manja; Ghosh, Pia M.; Madison, Cindee. - : Oxford University Press, 2013 BASE Show details

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