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Data for: Measuring Concept Semantic Relatedness through Common Spatial Pattern Feature Extraction on EEG Signals ...
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Atypical cortical entrainment to speech in the right hemisphere underpins phonemic deficits in dyslexia. ...
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Neural Encoding of Attended Continuous Speech under Different Types of Interference. ...
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Exploratory accross-stimulus studies in event-related potentials
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Neural Encoding of Attended Continuous Speech under Different Types of Interference.
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Atypical cortical entrainment to speech in the right hemisphere underpins phonemic deficits in dyslexia.
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Bilateral Auditory Development and Function in Children with Asymmetric Hearing Loss Who Listen with Electric and Acoustic Hearing
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Atypical cortical entrainment to speech in the right hemisphere underpins phonemic deficits in dyslexia
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| 90 |
The influence of a first language : training nonnative listeners on voicing contrasts
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One way or another : evidence for perceptual asymmetry in pre-attentive learning of non-native contrasts
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Effects of lexical ambiguity on perception: A test of the label feedback hypothesis using a visual oddball paradigm.
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In: Brain and Mind Institute Researchers' Publications (2018)
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ERP evidence of distinct processes underlying semantic facilitation and interference in word production.
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In: Cortex; a journal devoted to the study of the nervous system and behavior, vol. 99, pp. 1-12 (2018)
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Behavioral and electrophysiological signatures of word translation processes.
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In: Neuropsychologia, vol. 109, pp. 245-254 (2018)
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What's what in auditory cortices?
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In: NeuroImage, vol. 176, pp. 29-40 (2018)
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MAPPING LANGUAGE FUNCTION AND PREDICTING CORTICAL STIMULATION RESULTS WITH INTRACRANIAL ELECTROENCEPHALOGRAPHY
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Role of N-methyl-D-aspartate receptors in action-based predictive coding deficits in schizophrenia
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Beyond bilingual advantages: contexts, mechanisms, and correlates of executive function in bilingual and monolingual children
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EEG power at 3 months in infants at high familial risk for autism
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Abstract:
BACKGROUND: Alterations in brain development during infancy may precede the behavioral manifestation of developmental disorders. Infants at increased risk for autism are also at increased risk for other developmental disorders, including, quite commonly, language disorders. Here we assess the extent to which electroencephalographic (EEG) differences in infants at high versus low familial risk for autism are present by 3 months of age, and elucidate the functional significance of EEG power at 3 months in predicting later development. METHODS: EEG data were acquired at 3 months in infant siblings of children with autism (high risk; n = 29) and infant siblings of typically developing children (low risk; n = 19) as part of a prospective, longitudinal investigation. Development across multiple domains was assessed at 6, 9, 12, 18, 24, and 36 months. Diagnosis of autism was determined at 18–36 months. We assessed relationships between 3-month-olds’ frontal EEG power and autism risk, autism outcome, language development, and development in other domains. RESULTS: Infants at high familial risk for autism had reduced frontal power at 3 months compared to infants at low familial risk for autism, across several frequency bands. Reduced frontal high-alpha power at 3 months was robustly associated with poorer expressive language at 12 months. CONCLUSIONS: Reduced frontal power at 3 months may indicate increased risk for reduced expressive language skills at 12 months. This finding aligns with prior studies suggesting reduced power is a marker for atypical brain function, and infants at familial risk for autism are also at increased risk for altered developmental functioning in non-autism-specific domains. ; Support for this work was provided by the National Institutes of Health (R01DC010290 to HTF and CAN; R21 DC 08637 to HTF), Autism Speaks (1323 to HTF), Simons Foundation (137186 to CAN), the American Brain Foundation (to ARL), Autism Science Foundation (to ARL), Nancy Lurie Marks Family Foundation (to ARL), and the American Australian Association (to KJV). The funding bodies did not have any role in the design, collection, analyses, and interpretation of data or in writing the manuscript. (R01DC010290 - National Institutes of Health; R21 DC 08637 - National Institutes of Health; 1323 - Autism Speaks; 137186 - Simons Foundation; American Brain Foundation; Autism Science Foundation; Nancy Lurie Marks Family Foundation; American Australian Association)
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Keyword:
Autism; Biomarker; Children; Clinical neurology; Cognition; Early development; Early intervention; Electroencephalography; Infant siblings; Language; Life sciences & biomedicine; Mechanisms; Neurosciences; Neurosciences & neurology; Outcomes; Psychology; Science & technology; Siblings research consortium; Spectrum disorder; Speech
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URL: https://doi.org/10.1186/s11689-017-9214-9
https://hdl.handle.net/2144/33288
http://gateway.webofknowledge.com/gateway/Gateway.cgi?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000410955700001&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=6e74115fe3da270499c3d65c9b17d654
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