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1
Message recovery after violation of predictable linguistic signal: an investigation into alternatives through mind rhyme ...
Starr, John. - : Open Science Framework, 2022
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2
Multidimensional apathy in behavioral variant frontotemporal dementia, primary progressive aphasia, and Alzheimer disease
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3
Supplemetary_Materials - Multidimensional Apathy in Behavioral Variant Frontotemporal Dementia, Primary Progressive Aphasia, and Alzheimer Disease ...
Radakovic, Ratko; Shuna Colville; Cranley, Denise. - : SAGE Journals, 2020
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4
Multidimensional Apathy in Behavioral Variant Frontotemporal Dementia, Primary Progressive Aphasia, and Alzheimer Disease ...
Radakovic, Ratko; Shuna Colville; Cranley, Denise. - : SAGE Journals, 2020
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5
Multidimensional Apathy in Behavioral Variant Frontotemporal Dementia, Primary Progressive Aphasia, and Alzheimer Disease ...
Radakovic, Ratko; Shuna Colville; Cranley, Denise. - : SAGE Journals, 2020
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6
Supplemetary_Materials - Multidimensional Apathy in Behavioral Variant Frontotemporal Dementia, Primary Progressive Aphasia, and Alzheimer Disease ...
Radakovic, Ratko; Shuna Colville; Cranley, Denise. - : SAGE Journals, 2020
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7
Multidimensional Apathy in Behavioral Variant Frontotemporal Dementia, Primary Progressive Aphasia, and Alzheimer Disease
In: J Geriatr Psychiatry Neurol (2020)
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8
Genome-wide association meta-analysis in 269,867 individuals identifies new genetic and functional links to intelligence.
In: Nature genetics, vol 50, iss 7 (2018)
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9
Bilingualism, social cognition and executive functions: A tale of chickens and eggs
Cox, Simon R.; Bak, Thomas H.; Allerhand, Michael. - : Pergamon Press, 2016
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10
Genetic Basis of a Cognitive Complexity Metric
Hansell, Narelle K.; Halford, Graeme S.; Andrews, Glenda. - : Public Library of Science, 2015
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11
Genetic basis of a cognitive complexity metric
Hansell, Narelle K.; Halford, Graeme S.; Andrews, Glenda. - : Public Library of Science, 2015
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12
The ENIGMA Consortium: large-scale collaborative analyses of neuroimaging and genetic data
In: ISSN: 1931-7557 ; EISSN: 1931-7565 ; Brain imaging and behavior (Brain Imaging Behav) ; https://hal-pasteur.archives-ouvertes.fr/pasteur-01967166 ; Brain imaging and behavior (Brain Imaging Behav), Secaucus, NJ : Springer, 2014, 8 (2), pp.152-182. ⟨10.1007/s11682-013-9269-5⟩ (2014)
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13
Associations between APOE and low-density lipoprotein cholesterol genotypes and cognitive and physical capability: the HALCyon programme
Alfred, Tamuno; Ben-Shlomo, Yoav; Cooper, Rachel. - : Springer Netherlands, 2014
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14
Childhood intelligence is heritable, highly polygenic and associated with FNBP1L
Benyamin, B.; Pourcain, Bst; Davis, O. S.. - : Nature Publishing Group, 2014
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15
Genetic Variants Influencing Biomarkers of Nutrition Are Not Associated with Cognitive Capability in Middle-Aged and Older Adults123
Alfred, Tamuno; Ben-Shlomo, Yoav; Cooper, Rachel. - : American Society for Nutrition, 2013
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16
Quantitative analysis of the Aramaic Qumran texts
Starr, John Michael. - : The University of Edinburgh, 2013
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17
Genetic Copy Number Variation and General Cognitive Ability
MacLeod, Andrew K.; Davies, Gail; Payton, Antony. - : Public Library of Science, 2012
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18
Genetic contributions to stability and change in intelligence from childhood to old age
Abstract: Understanding the determinants of healthy mental ageing is a priority for society today1,2. So far, we know that intelligence differences show high stability from childhood to old age3,4 and there are estimates of the genetic contribution to intelligence at different ages5,6. However, attempts to discover whether genetic causes contribute to differences in cognitive ageing have been relatively uninformative7–10. Here we provide an estimate of the genetic and environmental contributions to stability and change in intelligence across most of the human lifetime. We used genome-wide single nucleotide polymorphism (SNP) data from 1,940 unrelated individuals whose intelligence was measured in childhood (age 11 years) and again in old age (age 65, 70 or 79 years)11,12. We use a statistical method that allows genetic (co)variance to be estimated from SNP data on unrelated individuals13–17. We estimate that causal genetic variants in linkage disequilibrium with common SNPs account for 0.24 of the variation in cognitive ability change from childhood to old age. Using bivariate analysis, we estimate a genetic correlation between intelligence at age 11 years and in old age of 0.62. These estimates, derived from rarely available data on lifetime cognitive measures, warrant the search for genetic causes of cognitive stability and change.
Keyword: 1000 General; Cognitive decline; Common SNPS explain; Large proportion; Later life
URL: https://espace.library.uq.edu.au/view/UQ:268426
https://espace.library.uq.edu.au/view/UQ:268426/UQ268426_OA.pdf
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19
Brain lesions, hypertension and cognitive ageing in the 1921 and 1936 Aberdeen birth cohorts
Murray, Alison D.; Staff, Roger T.; McNeil, Chris J.. - : Springer Netherlands, 2011
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20
Genome-wide association studies establish that human intelligence is highly heritable and polygenic
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